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Structural Physiology of the Cryptosporidium Oocyst Wall - Ward, H Bhat, N O'Connora, R
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Ward, H Bhat, N O'Connora, R:
Structural Physiology of the Cryptosporidium Oocyst Wall - Livres de poche

2009, ISBN: 9781843399056

Edition reliée, ID: 830671990

Good., New India Publishing Agency, 2009. Hardcover. New. Plant Disease Management requires a basic knowledge about the disease causing organism, whether fungi, bacteria, viruses, Mycoplasma or any other microorganisms. In this book `Illustrated Plant Pathology--Basic Concepts`, the authors have covered: Detailed account of the various pathogenic microorganisms responsible for causing diseases. Their classification, life cycle, mode of survival, spread and infection, factors responsible for epidemics, physiological specialization of pathogens. Disease surveillance, assessment of disease intensity, methods of disease control, plant protection chemicals, plant protection appliances and other aspects, which may go a long way in adopting suitable measures to combat the diseases. Management of seed-borne, soil borne and foliar diseases, use of plant products in disease management and biological approaches in disease management have also been dealt with. Numerous illustrations have also been given to make the text easily understandable. Though intended for the students of Agriculture, the book will be highly useful for the people working in the Department of agriculture and to the elite public who are interested in scientific agriculture. Contents Foreword Preface List of Figures List of Tables Chapter _\“ 1 : Fundamentals of Plant Pathology Chapter _\“ 2 : Principles of Plant Disease Management References Glossary of Scientific Terms Subject Index Printed Pages: 492., New India Publishing Agency, 2009, AwwaRF, 2005. AwwaRF 2005 Fine/ Book DescriptionThe apicomplexan parasite Cryptosporidium parvum is a significant cause of human and animal diarrheal disease worldwide. This parasite is currently recognized as the causative agent of numerous outbreaks of waterborne diarrheal disease. C. parvum infection in immunocompetent individuals is asymptomatic or associated with self-limiting diarrheal illness. However in immunocompromised hosts, such as patients with acquired immunodeficiency syndrome (AIDS), Cryptosporidium may cause severe, protracted and possibly fatal diarrheal disease. C. parvum isolates can be divided into two genetically distinct groups, one designated genotype I, exclusively associated with human infections, and the other genotype II, associated with both human and animal infections. The majority of infections associated with waterborne outbreaks are of genotype I. Published genotypic information of C. parvum from waterborne outbreaks particularly in the USA suggests that up to 80% of infected humans excrete genotype I oocysts. However, most studies related to water borne transmission use genotype II oocysts. C. parvum oocysts can survive for many months in water and are resistant to several disinfectant treatments. The prolonged survival of oocysts as well as the resistance to disinfectants is attributed to the presence of a thick wall that is believed to serve a protective function by isolating the parasite from the external environment. Ultrastructurally, the oocyst wall consists of two electron dense layers, an outer irregular 10 nm layer separated by an electron-lucent space from an inner thicker electron dense layer. A distinctive feature of the oocyst wall is the presence of a suture spanning part of the circumference of the inner wall, which undergoes dissolution during excystation. Oocyst wall formation in Cryptosporidium is initiated in wall forming bodies present in macrogametes. Although the ultrastructural features of the oocyst wall and suture have been described in some detail, very little is known about the biochemical composition and structural physiology of these important structures. In addition, very little is known about the effect of various water treatment processes or disinfectants on individual components of the oocyst wall. The integrity of the oocyst wall is responsible for prolonged survival of C. parvum in drinking water sources as well as its resistance to various disinfectants. The biochemical composition of specific components, which contribute to the structural integrity of the Cryptosporidium oocyst wall, and the effect of water treatment and purification processes on them are largely unknown. Knowledge of these components is therefore crucial in designing strategies directed at detecting and eliminating C. parvum from drinking water supplies.. Paperback. Fine., AwwaRF, 2005

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Structural Physiology of the Cryptosporidium Oocyst Wall - H. Ward, N. Bhat, R. O'Connor, S. Jaison, G. Widmer, G. Batzer, B. Corey, B. Stein
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H. Ward, N. Bhat, R. O'Connor, S. Jaison, G. Widmer, G. Batzer, B. Corey, B. Stein:
Structural Physiology of the Cryptosporidium Oocyst Wall - Livres de poche

2007, ISBN: 1843399059

Paperback, [EAN: 9781843399056], Iwa Publishing, Iwa Publishing, Book, [PU: Iwa Publishing], 2005-04-27, Iwa Publishing, 2205888011, Custom Stores, 7187330011, Amazon Publishing, 13901671, Audiobooks, 14244971, Bargain Books, 13621591, Boxed Sets, 194725011, Canada Day Deals, 377366011, Christian Living Store, 263003011, For Dummies Store, 2354164011, Formats, 714830011, Kids & Family Store, 370821011, Qualifying Textbooks - Fall 2007, 1040638, Specialty Stores, 916520, Books, 948352, Biochemistry, 948348, Basic Sciences, 948300, Medical Books, 927726, Subjects, 916520, Books, 948458, Communicable, 948408, Diseases, 948670, Pathology, 948734, Internal Medicine, 951884, Medicine, 948300, Medical Books, 927726, Subjects, 916520, Books, 948750, Infectious Disease, 948458, Communicable Diseases, 948754, Parasitology, 948756, Tropical Medicine, 948734, Internal Medicine, 951884, Medicine, 948300, Medical Books, 927726, Subjects, 916520, Books, 952068, Biochemistry, 952052, Agricultural Sciences, 952050, Professional Science, 950756, Professional & Technical, 927726, Subjects, 916520, Books, 952258, Environmental Science, 952256, Earth Sciences, 952050, Professional Science, 950756, Professional & Technical, 927726, Subjects, 916520, Books, 387101011, Environmental, 387071011, Civil, 387057011, Engineering, 950756, Professional & Technical, 927726, Subjects, 916520, Books, 950618, Environmental Science, 950604, Environment, 956280, Science & Math, 927726, Subjects, 916520, Books, 956820, Biology, 956824, Cell Biology, 956828, Developmental Biology, 956830, Entomology, 51331011, Extraterrestrial, 51332011, Freshwater Biology, 956566, Marine Biology, 956826, Microbiology, 956370, Molecular Biology, 51333011, Systematics, 956812, Biological Sciences, 956280, Science & Math, 927726, Subjects, 916520, Books, 956302, Biochemistry, 956282, Chemistry, 956280, Science & Math, 927726, Subjects, 916520, Books, 956310, Environmental Science, 956306, Earth Sciences, 956280, Science & Math, 927726, Subjects, 916520, Books, 956920, Biochemistry, 956916, Basic Science, 956868, Medicine, 956280, Science & Math, 927726, Subjects, 916520, Books, 957026, Communicable, 956976, Diseases, 956868, Medicine, 956280, Science & Math, 927726, Subjects, 916520, Books, 957306, Infectious Disease, 957026, Communicable Diseases, 957308, Epidemiology, 957310, Parasitology, 957312, Tropical Medicine, 957290, Internal Medicine, 956868, Medicine, 956280, Science & Math, 927726, Subjects, 916520, Books, 956788, Technology, 956790, Futurology, 956408, History of Technology, 106448011, Innovations, 956794, Nanotechnology, 106449011, Philosophy of Technology, 956800, Renewable Energy, 956796, Risks, 956798, Safety & Health, 274725011, Social Aspects, 956802, Technical Thinking & Writing, 106450011, Technology & Public Policy, 106451011, Technology & Society, 106452011, Technology Transfer, 956280, Science & Math, 927726, Subjects, 916520, Books, 690358011, Environmental Engineering, 15305951, Engineering, 15115321, Textbooks, 927726, Subjects, 916520, Books, 690426011, Biochemistry, 690424011, Basic Sciences, 690423011, Medicine, 15306671, Medicine, 15115321, Textbooks, 927726, Subjects, 916520, Books, 690458011, Infectious Diseases, 690440011, Clinical, 690423011, Medicine, 15306671, Medicine, 15115321, Textbooks, 927726, Subjects, 916520, Books, 690544011, Biology, 15306931, Biology & Life Sciences, 15306801, Sciences, 15115321, Textbooks, 927726, Subjects, 916520, Books, 690542011, Environmental Studies, 15306801, Sciences, 15115321, Textbooks, 927726, Subjects, 916520, Books

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Structural Physiology of the Cryptosporidium Oocyst Wall - Ward, H Bhat, N O'Connora, R
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Structural Physiology of the Cryptosporidium Oocyst Wall - exemplaire signée

2005, ISBN: 9781843399056

Livres de poche, Edition reliée, ID: 44785777

London: Free Press of Glencoe. 1963. First. First edition. Very good in good dustwrapper. Hardcover lightly rubbed and bumped at spine ends and corners. Dustwrapper yellowed, worn along edges and corners. Inscribed on front end page by editor. ., Free Press of Glencoe, 1963, AwwaRF, 2005. AwwaRF 2005 Fine/ Book DescriptionThe apicomplexan parasite Cryptosporidium parvum is a significant cause of human and animal diarrheal disease worldwide. This parasite is currently recognized as the causative agent of numerous outbreaks of waterborne diarrheal disease. C. parvum infection in immunocompetent individuals is asymptomatic or associated with self-limiting diarrheal illness. However in immunocompromised hosts, such as patients with acquired immunodeficiency syndrome (AIDS), Cryptosporidium may cause severe, protracted and possibly fatal diarrheal disease. C. parvum isolates can be divided into two genetically distinct groups, one designated genotype I, exclusively associated with human infections, and the other genotype II, associated with both human and animal infections. The majority of infections associated with waterborne outbreaks are of genotype I. Published genotypic information of C. parvum from waterborne outbreaks particularly in the USA suggests that up to 80% of infected humans excrete genotype I oocysts. However, most studies related to water borne transmission use genotype II oocysts. C. parvum oocysts can survive for many months in water and are resistant to several disinfectant treatments. The prolonged survival of oocysts as well as the resistance to disinfectants is attributed to the presence of a thick wall that is believed to serve a protective function by isolating the parasite from the external environment. Ultrastructurally, the oocyst wall consists of two electron dense layers, an outer irregular 10 nm layer separated by an electron-lucent space from an inner thicker electron dense layer. A distinctive feature of the oocyst wall is the presence of a suture spanning part of the circumference of the inner wall, which undergoes dissolution during excystation. Oocyst wall formation in Cryptosporidium is initiated in wall forming bodies present in macrogametes. Although the ultrastructural features of the oocyst wall and suture have been described in some detail, very little is known about the biochemical composition and structural physiology of these important structures. In addition, very little is known about the effect of various water treatment processes or disinfectants on individual components of the oocyst wall. The integrity of the oocyst wall is responsible for prolonged survival of C. parvum in drinking water sources as well as its resistance to various disinfectants. The biochemical composition of specific components, which contribute to the structural integrity of the Cryptosporidium oocyst wall, and the effect of water treatment and purification processes on them are largely unknown. Knowledge of these components is therefore crucial in designing strategies directed at detecting and eliminating C. parvum from drinking water supplies.. Paperback. Fine., AwwaRF, 2005

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Structural Physiology of the Cryptosporidium Oocyst Wall - Ward, H Bhat, N O'Connora, R
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Ward, H Bhat, N O'Connora, R:
Structural Physiology of the Cryptosporidium Oocyst Wall - Livres de poche

2005, ISBN: 1843399059

ID: 1013932022

[EAN: 9781843399056], Fine, [PU: AwwaRF], AWWA RESEARCH FOUNDATION REPORTS, AwwaRF 2005 Fine/ Book DescriptionThe apicomplexan parasite Cryptosporidium parvum is a significant cause of human and animal diarrheal disease worldwide. This parasite is currently recognized as the causative agent of numerous outbreaks of waterborne diarrheal disease. C. parvum infection in immunocompetent individuals is asymptomatic or associated with self-limiting diarrheal illness. However in immunocompromised hosts, such as patients with acquired immunodeficiency syndrome (AIDS), Cryptosporidium may cause severe, protracted and possibly fatal diarrheal disease. C. parvum isolates can be divided into two genetically distinct groups, one designated genotype I, exclusively associated with human infections, and the other genotype II, associated with both human and animal infections. The majority of infections associated with waterborne outbreaks are of genotype I. Published genotypic information of C. parvum from waterborne outbreaks particularly in the USA suggests that up to 80% of infected humans excrete genotype I oocysts. However, most studies related to water borne transmission use genotype II oocysts. C. parvum oocysts can survive for many months in water and are resistant to several disinfectant treatments. The prolonged survival of oocysts as well as the resistance to disinfectants is attributed to the presence of a thick wall that is believed to serve a protective function by isolating the parasite from the external environment. Ultrastructurally, the oocyst wall consists of two electron dense layers, an outer irregular 10 nm layer separated by an electron-lucent space from an inner thicker electron dense layer. A distinctive feature of the oocyst wall is the presence of a suture spanning part of the circumference of the inner wall, which undergoes dissolution during excystation. Oocyst wall formation in Cryptosporidium is initiated in wall forming bodies present in macrogametes. Although the ultrastructural features of the oocyst wall and suture have been described in some detail, very little is known about the biochemical composition and structural physiology of these important structures. In addition, very little is known about the effect of various water treatment processes or disinfectants on individual components of the oocyst wall. The integrity of the oocyst wall is responsible for prolonged survival of C. parvum in drinking water sources as well as its resistance to various disinfectants. The biochemical composition of specific components, which contribute to the structural integrity of the Cryptosporidium oocyst wall, and the effect of water treatment and purification processes on them are largely unknown. Knowledge of these components is therefore crucial in designing strategies directed at detecting and eliminating C. parvum from drinking water supplies.

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Structural Physiology Of The Cryptosporidium Oocyst Wall
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Structural Physiology Of The Cryptosporidium Oocyst Wall - nouveau livre

ISBN: 9781843399056

ID: 9644579

The apicomplexan parasite Cryptosporidium parvum is a significant cause of human and animal diarrheal disease worldwide. This parasite is currently recognized as the causative agent of numerous outbreaks of waterborne diarrheal disease. C. parvum infection in immunocompetent individuals is asymptomatic or associated with self-limiting diarrheal illness. However in immunocompromised hosts, such. The apicomplexan parasite Cryptosporidium parvum is a significant cause of human and animal diarrheal disease worldwide. This parasite is currently recognized as the causative agent of numerous outbreaks of waterborne diarrheal disease. C. parvum infection in immunocompetent individuals is asymptomatic or associated with self-limiting diarrheal illness. However in immunocompromised hosts, such as patients with acquired immunodeficiency syndrome (AIDS), Cryptosporidium may cause severe, protracted and possibly fatal diarrheal disease. C. parvum isolates can be divided into two genetically distinct groups, one designated genotype I, exclusively associated with human infections, and the other genotype II, associated with both human and animal infections. The majority of infections associated with waterborne outbreaks are of genotype I. Published genotypic information of C. parvum from waterborne outbreaks particularly in the USA suggests that up to 80 per cent of infected humans excrete genotype I oocysts. However, most studies related to water borne transmission USE genotype II oocysts. C. parvum oocysts can survive for many months in water and are resistant to several disinfectant treatments. The prolonged survival of oocysts as well as the resistance to disinfectants is attributed to the presence of a thick wall that is believed to serve a protective function by isolating the parasite from the external environment. Ultrastructurally, the oocyst wall consists of two electron dense layers, an outer irregular 10 nm layer separated by an electron-lucent space from an inner thicker electron dense layer. A distinctive feature of the oocyst wall is the presence of a suture spanning part of the circumference of the inner wall, which undergoes dissolution during excystation. Oocyst wall formation in Cryptosporidium is initiated in wall forming bodies present in macrogametes. Although the ultrastructural features of the oocyst wall and suture have been described in some. Books, Medical~~Clinical & Internal Medicine~~Diseases & Disorders, Structural Physiology Of The Cryptosporidium Oocyst Wall~~Book~~9781843399056~~H Ward, N Bhat, R O'Connora, , , , , , , , , ,

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Structural Physiology of the Cryptosporidium Oocyst Wall

The apicomplexan parasite Cryptosporidium parvum is a significant cause of human and animal diarrheal disease worldwide. This parasite is currently recognized as the causative agent of numerous outbreaks of waterborne diarrheal disease. C. parvum infection in immunocompetent individuals is asymptomatic or associated with self-limiting diarrheal illness. However in immunocompromised hosts, such as patients with acquired immunodeficiency syndrome (AIDS), Cryptosporidium may cause severe, protracted and possibly fatal diarrheal disease. C. parvum isolates can be divided into two genetically distinct groups, one designated genotype I, exclusively associated with human infections, and the other genotype II, associated with both human and animal infections. The majority of infections associated with waterborne outbreaks are of genotype I. Published genotypic information of C. parvum from waterborne outbreaks particularly in the USA suggests that up to 80 per cent of infected humans excrete genotype I oocysts. However, most studies related to water borne transmission use genotype II oocysts. C. parvum oocysts can survive for many months in water and are resistant to several disinfectant treatments. The prolonged survival of oocysts as well as the resistance to disinfectants is attributed to the presence of a thick wall that is believed to serve a protective function by isolating the parasite from the external environment. Ultrastructurally, the oocyst wall consists of two electron dense layers, an outer irregular 10 nm layer separated by an electron-lucent space from an inner thicker electron dense layer. A distinctive feature of the oocyst wall is the presence of a suture spanning part of the circumference of the inner wall, which undergoes dissolution during excystation. Oocyst wall formation in Cryptosporidium is initiated in wall forming bodies present in macrogametes. Although the ultrastructural features of the oocyst wall and suture have been described in some detail, very little is known about the biochemical composition and structural physiology of these important structures. In addition, very little is known about the effect of various water treatment processes or disinfectants on individual components of the oocyst wall. The integrity of the oocyst wall is responsible for prolonged survival of C. parvum in drinking water sources as well as its resistance to various disinfectants. The biochemical composition of specific components, which contribute to the structural integrity of the Cryptosporidium oocyst wall, and the effect of water treatment and purification processes on them are largely unknown. Knowledge of these components is therefore crucial in designing strategies directed at detecting and eliminating C. parvum from drinking water supplies.

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EAN (ISBN-13): 9781843399056
ISBN (ISBN-10): 1843399059
Version reliée
Livre de poche
Date de parution: 2005
Editeur: AWWARF
88 Pages
Poids: 0,272 kg
Langue: eng/Englisch

Livre dans la base de données depuis 07.11.2007 09:04:09
Livre trouvé récemment le 27.02.2017 10:49:54
ISBN/EAN: 1843399059

ISBN - Autres types d'écriture:
1-84339-905-9, 978-1-84339-905-6


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